What is the result of cholecystokinin (CCK) stimulation on the gallbladder?

Cholecystokinin (CCK) plays a critical role in the digestion of fats and proteins by influencing the gallbladder's function. When CCK is released, typically in response to the presence of fats and proteins in the small intestine, it stimulates the gallbladder to contract. This contraction is crucial for the expulsion of bile, which is necessary for the emulsification and digestion of fats.

The increased contractility of the gallbladder facilitated by CCK leads to the release of bile into the duodenum, supporting the digestive process. This action helps ensure that dietary fats are effectively broken down and absorbed.

In contrast, the other options involve mechanisms that are not aligned with the physiological effects of CCK. For example, decreased contractility would impede bile release, inhibiting digestion rather than aiding it. Similarly, CCK enhances bile production but does not inhibit it. The relaxation of the sphincter of Oddi is also an action stimulated by CCK, but the primary effect regarding the gallbladder is its contraction, leading to increased contractility and subsequent bile release. Thus, the understanding of CCK's role ultimately reinforces why increased contractility is the correct outcome concerning the gallbladder’s response to this hormone